In a new study by University of Vanderbilt shows a experimental molecule that causes kidney failure in mosquitos which leads them to explode.
Vanderbilt pharmacologist Jerod Denton, Ph.D., Ohio State entomologist Peter Piermarini, Ph.D., and colleagues have aimed their study's at Anopheles gambiae which is the main cause of malaria in the world, and also Aedes aegypti which is the main transmitter of the Zika virus. Over decades mosquitoes evolve more and more to genetic resistance to insecticides which target their immune system. The study shows kidney failure, but not just kidney failure Malpighi an tubule failure.
"We're essentially preventing mosquitoes from producing urine after they take a blood meal," said Denton, associate professor of Anesthesiology and Pharmacology.
When a mosquito takes a blood meal their body weight at least doubles or triples in size. Besides providing nutrients to mosquitos blood meals also carry toxic salts which can depolarize cell membrane if not quickly voided. So this is all a rapid process to the mosquito. What the compound does would stop urine production the mosquito. After this happens the mosquito would swell up and burst. However this is only targeting the blood feeding mosquitos which are females.
"By targeting blood feeding female mosquitoes, we predict that there will be less selective pressure for the emergence of resistant mutations," Denton said
I believe this compound could greatly help in regulating mosquitos. It would also decrease the risk of malaria, Zika and other viruses.